A study published in the journal Neuron has found the cellular mechanism that links stress in the brain to the progression of Alzheimer's disease. This breakthrough can promise a major boost in the treatment of the disease.
Researchers with the Advanced Science Research Center at the CUNY Graduate Center (CUNY ASRC) have highlighted microglia, the brain's primary immune cells, as central players in both the protective and harmful responses associated with Alzheimer’s disease, which is a progressive neurodegenerative disorder that primarily affects memory, thinking, and behavior. It is the most common cause of dementia, often beginning with mild memory loss and advancing to severe cognitive and functional impairment. The condition is characterized by the accumulation of amyloid plaques and tau tangles in the brain, leading to neuronal damage and brain shrinkage.
